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- ...009) [http://dx.doi.org/10.1016/j.mehy.2009.04.047 Permanent impairment of insulin resistance from pregnancy to adulthood: the primary basic risk factor of ch ...k factors associated with IR have to be taken into account.</font><!--\Dat\Insulin Resistance\-->4 KB (600 words) - 17:05, 25 February 2013
- {{r|Insulin}} {{r|Insulin-like growth factor}} (IGF, also somatomedin)1 KB (175 words) - 08:13, 8 November 2010
- ...rease in response to a variety of hormones, including [[acetylcholine]], [[insulin]], and [[oxytocin]] and it has been found to activate specific protein [[ki385 bytes (52 words) - 17:06, 14 May 2010
- ...molecules are added to chains of glycogen. This process is activated by [[insulin]] in response to high glucose levels. ...ynthase is activated by phosphoprotein phoshatase, which is activated by [[insulin]].1 KB (211 words) - 09:00, 14 September 2013
- See ''Article on [[insulin]], if necessary, to better understand present article.'' ...the 2013 Medical Subject Headings of the National Library of Medicine, '''insulin resistance''' consists of8 KB (1,179 words) - 17:37, 10 March 2014
- ...lphia General Hospital and in 1927 he submitted a thesis on "The Sulfur in Insulin" to the School of Medicine of [[Rochester University]] which earned him the ...hns Hopkins University Medical School]], where he continued his studies on insulin. A second fellowship enabled him to travel abroad, and work with George Bar2 KB (368 words) - 16:47, 27 January 2023
- ...useful quantities of a chemical manufactured by the human body, such as [[insulin]]. ...ular modifications were made to change the duration of action of a dose of insulin, while still having the same role in sugar metabolism, it can be argued if5 KB (817 words) - 06:29, 31 May 2009
- {{r|Insulin resistance}}491 bytes (62 words) - 15:57, 11 January 2010
- ...bdominal fat; atherogenic dyslipidemia; [[hypertension]]; hyperglycemia; [[insulin resistance]]; a proinflammatory state; and a prothrombotic (thrombosis) sta629 bytes (71 words) - 11:35, 17 April 2009
- ...e]]. Glucose molecules are added to the chains of glycogen as long as both insulin and glucose remain plentiful. In this [[postprandial]] or "fed" state, the After a meal has been digested and glucose levels begin to fall, insulin secretion is reduced, and glycogen synthesis stops. About four hours after5 KB (834 words) - 14:45, 26 September 2007
- {{r|Insulin-secreting cell}} {{r|Insulin}}2 KB (302 words) - 14:21, 8 March 2024
- {{r|Insulin}}619 bytes (77 words) - 18:53, 11 January 2010
- *[[IGF-1]] and other [[insulin-like growth factor]]s *[[Insulin]]2 KB (286 words) - 02:03, 2 June 2009
- {{r|Insulin}}520 bytes (65 words) - 16:52, 11 January 2010
- Auto-populated based on [[Special:WhatLinksHere/Insulin resistance]]. Needs checking by a human.665 bytes (82 words) - 17:27, 11 January 2010
- {{r|Insulin resistance}}692 bytes (87 words) - 18:27, 11 January 2010
- {{r|Insulin}}605 bytes (75 words) - 15:56, 11 January 2010
- {{r|Insulin}}864 bytes (108 words) - 15:56, 11 January 2010
- ....'' (2003) The atherogenic lipoprotein profile associated with obesity and insulin resistance is largely attributable to intra-abdominal fat. ''Diabetes'' 52: ..., Clapham JC (1998) Insulin resistance, impaired glucose tolerance and non-insulin dependent diabetes, pathologic mechanism and treatment: current status and5 KB (669 words) - 08:49, 11 October 2010
- *[[Insulin]], produced in the pancreas {{r|Insulin-like growth factor}}2 KB (295 words) - 10:18, 30 May 2009