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  • ...'''cangrelor''' is an [[purinergic P2Y receptor antagonist]]s that is a [[platelet aggregation inhibitor]]. ...Stone GW, Mahaffey KW, Gibson CM, Steg PG, Hamm CW et al.| title=Effect of platelet inhibition with cangrelor during PCI on ischemic events. | journal=N Engl J
    6 KB (873 words) - 12:25, 27 August 2013
  • {{r|Platelet}}
    828 bytes (107 words) - 22:30, 17 April 2011
  • {{r|Platelet transfusion}}
    816 bytes (122 words) - 01:41, 15 November 2010
  • ...antagonize or impair any mechanism leading to blood [[platelet activation|platelet aggregation]], whether during the phases of activation and shape change or Inhibitors of the [[platelet glycoprotein GPIIb-IIIa complex]] (GPIs) include the [[monoclonal antibody]
    4 KB (543 words) - 01:43, 8 May 2013
  • {{r|Platelet}}
    1 KB (147 words) - 07:44, 8 January 2010
  • {{r|Platelet}}
    1 KB (146 words) - 21:45, 23 August 2010
  • ==Anatomy of a platelet== ...brand factor]]), the contents of which are released upon activation of the platelet. These granule contents play an important role in both [[hemostasis]] and i
    12 KB (1,658 words) - 08:52, 28 June 2011
  • ...nce of [[fibrin]] strands following the mixing of plasma with phospholipid platelet substitute (e.g., crude cephalins, soybean phosphatides). It is a test of t
    1 KB (155 words) - 10:43, 23 August 2010
  • ...nism of [[phagocytosis]]. They produce [[leukotriene]] C<sub>4</sub> and [[platelet activation factor]] (PAF). <ref name=Ganong>{{citation ...Ganong, p. 508</ref> Especially infectious parasites produce inhibitors of platelet aggregation.<ref name=Liu>{{citation
    4 KB (579 words) - 22:42, 9 June 2010
  • ...of [[hematoepoesis]]: generation of [[erythrocyte]]s, [[leukocyte]]s and [[platelet]]s from [[hematopoetic stem cell]]s.
    1 KB (202 words) - 20:28, 25 June 2010
  • {{r|Platelet||**}}
    2 KB (192 words) - 08:17, 11 December 2009
  • {{r|Platelet-derived growth factor receptor||**}}
    2 KB (234 words) - 00:42, 13 January 2009
  • ...nthase]] by acetylating its terminal amino group. Aspirin also inhibits [[platelet]] aggregation and is used in the prevention of arterial and venous [[thromb Individuals in whom platelet function assays demonstrate resistance to aspirin are less likely to have c
    4 KB (523 words) - 14:17, 26 July 2017
  • ...s]] include [[leukocyte]]s for immune and infection-fighting action, and [[platelet]]s for clotting.
    2 KB (261 words) - 09:00, 1 March 2024
  • ...eptor]]s, and [[purinoceptor P2Y12|purinoceptor P2Y<sub>12</sub>]] (causes platelet aggregation).
    3 KB (338 words) - 13:08, 30 March 2010
  • *[[Platelet]]s per unit volume
    2 KB (257 words) - 12:46, 30 January 2011
  • ...ansduction pathways; for example, protein kinase activation is part of the platelet activation signal pathway."<ref>{{MeSH}}</ref>
    2 KB (329 words) - 10:52, 9 July 2009
  • ...('''TTP''') is "an acquired, congenital, or familial disorder caused by [[platelet aggregation]] with [[thrombosis]] in terminal arterioles and capillaries. C
    2 KB (310 words) - 21:03, 25 September 2013
  • ...hich leads to profound low [[white blood cell]], [[red blood cell]], and [[platelet]] counts.
    2 KB (280 words) - 23:21, 10 June 2010
  • ...does not irreversibly inhibit [[cyclooxygenase]] and thus is not an [[anti-platelet agent]].
    2 KB (338 words) - 13:15, 22 November 2011
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